Nuclear Interleukin-33 Is Generally Expressed in Resting Endothelium but Rapidly Lost upon Angiogenic or Proinflammatory Activation
- 31 October 2008
- journal article
- Published by Elsevier BV in The American Journal of Pathology
- Vol. 173 (4), 1229-1242
- https://doi.org/10.2353/ajpath.2008.080014
Abstract
No abstract availableKeywords
This publication has 49 references indexed in Scilit:
- Induction of IL-33 expression and activity in central nervous system gliaJournal of Leukocyte Biology, 2008
- A novel IL-1 family cytokine, IL-33, potently activates human eosinophilsJournal of Allergy and Clinical Immunology, 2008
- IL-33 reduces the development of atherosclerosisThe Journal of Experimental Medicine, 2008
- IL-1 receptor accessory protein is essential for IL-33-induced activation of T lymphocytes and mast cellsProceedings of the National Academy of Sciences of the United States of America, 2007
- IL-33 and ST2 comprise a critical biomechanically induced and cardioprotective signaling systemJCI Insight, 2007
- IL-33, the IL-1-like cytokine ligand for ST2 receptor, is a chromatin-associated nuclear factorin vivoProceedings of the National Academy of Sciences, 2007
- Inflammation and cancerNature, 2002
- Selective inhibition of endothelial cell proliferation by fumagillin is not due to differential expression of methionine aminopeptidasesJournal of Cellular Biochemistry, 2000
- Abnormal blood vessel development and lethality in embryos lacking a single VEGF alleleNature, 1996
- Tumors: Wounds That Do Not HealNew England Journal of Medicine, 1986