Akt Activation Is Responsible for Enhanced Migratory and Invasive Behavior of Arsenic-Transformed Human Bronchial Epithelial Cells
- 1 January 2012
- journal article
- Published by Environmental Health Perspectives in Environmental Health Perspectives
- Vol. 120 (1), 92-97
- https://doi.org/10.1289/ehp.1104061
Abstract
A peer-reviewed open access journal published by the National Institute of Environmental Health SciencesKeywords
This publication has 36 references indexed in Scilit:
- Invasiveness and anchorage independent growth ability augmented by PTEN inactivation through the PI3K/AKT/NFkB pathway in lung cancer cellsLung Cancer, 2011
- Reduced Reactive Oxygen Species–Generating Capacity Contributes to the Enhanced Cell Growth of Arsenic-Transformed Epithelial CellsCancer Research, 2010
- Mechanisms of non-genotoxic carcinogens and importance of a weight of evidence approachMutation Research - Reviews in Mutation Research, 2009
- Epigenetics in metal carcinogenesis: nickel, arsenic, chromium and cadmiumMetallomics, 2009
- Arsenic in drinking water and lung cancer: A systematic reviewEnvironmental Research, 2008
- Inorganic Arsenic and Human Prostate CancerEnvironmental Health Perspectives, 2008
- Mitogenic Signal Transduction Caused by Monomethylarsonous Acid in Human Bladder Cells: Role in Arsenic-Induced CarcinogenesisToxicological Sciences, 2006
- Perturbations of the AKT signaling pathway in human cancerOncogene, 2005
- Inorganic Arsenite-Induced Malignant Transformation of Human Prostate Epithelial CellsJNCI Journal of the National Cancer Institute, 2002
- Requirement of Erk, but Not JNK, for Arsenite-induced Cell TransformationOnline Journal of Public Health Informatics, 1999