Requirement of Erk, but Not JNK, for Arsenite-induced Cell Transformation
Open Access
- 1 May 1999
- journal article
- research article
- Published by Elsevier BV
- Vol. 274 (21), 14595-14601
- https://doi.org/10.1074/jbc.274.21.14595
Abstract
No abstract availableThis publication has 37 references indexed in Scilit:
- The absorption of arsenic and its relation to carcinomaBritish Journal of Dermatology, 2006
- Expression of dominant negative Erk2 inhibits AP-1 transactivation and neoplastic transformationOncogene, 1998
- Increased synthesis of phosphocholine is required for UV-induced AP-1 activationOncogene, 1998
- Regulation of DNA binding and transactivation in p53 by nuclear localization and phosphorylationOncogene, 1997
- Differential activation of ERK, JNK/SAPK and P3/CSBP/RK map kinase family members during the cellular response to arseniteFree Radical Biology & Medicine, 1996
- Ultraviolet B-induced Activated Protein-1 Activation Does Not Require Epidermal Growth Factor Receptor but Is Blocked by a Dominant Negative PKCλ/ιPublished by Elsevier BV ,1996
- Physiological and Toxicological Changes in the Skin Resulting from the Action and Interaction of Metal IonsCritical Reviews in Toxicology, 1995
- Genotoxic effects of sodium arsenite on human cellsMutation Research, 1992
- Tumour promoter induces anchorage independence irreversiblyNature, 1979
- Arsenic and cancerBritish Journal of Dermatology, 1977