Reperfusion Injury as the Mechanism of Brain Damage after Perinatal Asphyxia
- 1 May 1997
- journal article
- review article
- Published by Springer Science and Business Media LLC in Pediatric Research
- Vol. 41 (5), 599-606
- https://doi.org/10.1203/00006450-199705000-00001
Abstract
No abstract availableKeywords
This publication has 122 references indexed in Scilit:
- Microglia activation after neonatal hypoxic-ischemiaDevelopmental Brain Research, 1995
- Purification and immunohistochemical tissue localization of human xanthine oxidaseBiochimica et Biophysica Acta (BBA) - Protein Structure and Molecular Enzymology, 1993
- Glutamate triggers internucleosomal DNA cleavage in neuronal cellsBiochemical and Biophysical Research Communications, 1991
- Cerebral blood flow, cross-brain oxygen extraction, and fontanelle pressure after hypoxic-ischemic injury in newborn infantsThe Journal of Pediatrics, 1991
- Vasoparalysis associated with brain damage in asphyxiated term infantsThe Journal of Pediatrics, 1990
- Tissue Destruction by NeutrophilsNew England Journal of Medicine, 1989
- Xanthine oxidase produces hydrogen peroxide which contributes to reperfusion injury of ischemic, isolated, perfused rat hearts.JCI Insight, 1988
- Reduction of the extent of ischemic myocardial injury by neutrophil depletion in the dog.Cell Metabolism, 1983
- Temporal profile of neuronal damage in a model of transient forebrain ischemiaAnnals of Neurology, 1982
- Uric acid provides an antioxidant defense in humans against oxidant- and radical-caused aging and cancer: a hypothesis.Proceedings of the National Academy of Sciences, 1981