Analysis of clonality of atypical cutaneous lymphoid infiltrates associated with drug therapy by PCR/DGGE
- 1 February 1999
- journal article
- Published by Elsevier BV in Human Pathology
- Vol. 30 (2), 130-136
- https://doi.org/10.1016/s0046-8177(99)90266-6
Abstract
No abstract availableKeywords
This publication has 20 references indexed in Scilit:
- DNA Extraction from Formalin-fixed, Paraffin-embedded TissuesDiagnostic Molecular Pathology, 1997
- Drug-induced immune dysregulation as a cause of atypical cutaneous lymphoid infiltrates: A hypothesisHuman Pathology, 1996
- Detection of clonal T-cell receptor gamma gene rearrangements with the use of the polymerase chain reaction in cutaneous lesions of mycosis fungoides and Sezary syndromeArchives of Dermatology, 1995
- Antidepressant therapy. A possible cause of atypical cutaneous lymphoid hyperplasiaArchives of Dermatology, 1995
- Drugs with antihistaminic properties as a cause of atypical cutaneous lymphoid hyperplasiaJournal of the American Academy of Dermatology, 1995
- Optimal primer selection for clonality assessment by polymerase chain reaction analysis: II. Follicular lymphomasHuman Pathology, 1994
- Optimal primer selection for clonality assessment by polymerase chain reaction analysis: I. Low grade B-cell lymphoproliferative disorders of nonfollicular center cell typeHuman Pathology, 1994
- Value of PCR Detection of TCR7 Gene Rearrangement in the Diagnosis ( Cutaneous Lymphocytic InfiltratesDiagnostic Molecular Pathology, 1994
- Detection of Clonal T-Cell Receptor γ Gene Rearrangements in Early Mycosis Fungoides/Sezary Syndrome by Polymerase Chain Reaction and Denaturing Gradient Gel Electrophoresis (PCR/DGGE)Journal of Investigative Dermatology, 1994
- Cutaneous lymphoproliferative disorders: strategies for molecular biological analysis and their major findingsSpringer Seminars in Immunopathology, 1992