NAD+-dependent SIRT1 Deacetylase Participates in Epigenetic Reprogramming during Endotoxin Tolerance
Open Access
- 1 March 2011
- journal article
- Published by Elsevier BV
- Vol. 286 (11), 9856-9864
- https://doi.org/10.1074/jbc.m110.196790
Abstract
No abstract availableKeywords
This publication has 38 references indexed in Scilit:
- The NF-κB Factor RelB and Histone H3 Lysine Methyltransferase G9a Directly Interact to Generate Epigenetic Silencing in Endotoxin TolerancePublished by Elsevier BV ,2009
- Transcriptional control of the inflammatory responseNature Reviews Immunology, 2009
- Recent progress in the biology and physiology of sirtuinsNature, 2009
- Circadian Control of the NAD + Salvage Pathway by CLOCK-SIRT1Science, 2009
- Chromatin-Specific Remodeling by HMGB1 and Linker Histone H1 Silences Proinflammatory Genes during Endotoxin ToleranceMolecular and Cellular Biology, 2009
- Intracellular NAD levels regulate tumor necrosis factor protein synthesis in a sirtuin-dependent mannerNature Medicine, 2009
- The NAD World: A New Systemic Regulatory Network for Metabolism and Aging—Sirt1, Systemic NAD Biosynthesis, and Their ImportanceCell Biochemistry and Biophysics, 2009
- RelB Sustains IκBα Expression during Endotoxin ToleranceClinical and Vaccine Immunology, 2009
- Nampt: linking NAD biology, metabolism and cancerTrends in Endocrinology & Metabolism, 2008
- G9a and HP1 Couple Histone and DNA Methylation to TNFα Transcription Silencing during Endotoxin TolerancePublished by Elsevier BV ,2008