Increased interferon-γ, interleukin-12p40 and IL-8 production in Propionibacterium acnes-treated peripheral blood mononuclear cells from patient with acne vulgaris: Host response but not bacterial species is the determinant factor of the disease
- 31 July 2009
- journal article
- Published by Elsevier BV in Journal of Dermatological Science
- Vol. 55 (1), 47-52
- https://doi.org/10.1016/j.jdermsci.2009.02.015
Abstract
No abstract availableKeywords
This publication has 18 references indexed in Scilit:
- Review of the Innate Immune Response in Acne vulgaris: Activation of Toll-Like Receptor 2 in Acne Triggers Inflammatory Cytokine ResponsesDermatology, 2005
- Nadifloxacin, an antiacne quinolone antimicrobial, inhibits the production of proinflammatory cytokines by human peripheral blood mononuclear cells and normal human keratinocytesJournal of Dermatological Science, 2005
- What is the pathogenesis of acne?Experimental Dermatology, 2005
- Induction of allergic inflammation by interleukin-18 in experimental animal modelsImmunological Reviews, 2004
- Propionibacterium acnes-Reactive T Helper-1 Cells in the Skin of Patients with Acne VulgarisJournal of Investigative Dermatology, 2003
- Inflammatory Events Are Involved in Acne Lesion InitiationJournal of Investigative Dermatology, 2003
- Therapy for Acne VulgarisThe New England Journal of Medicine, 1997
- Activity of type 1 5α–reductase is greater in the follicular infrainfundibulum compared with the epidermisBritish Journal of Dermatology, 1997
- The influence of tetracyclines on T cell activationClinical and Experimental Immunology, 1995
- Monoclonal Antibody Labeling for Cytokeratins and Filaggrin in the Human Pilosebaceous Unit of Normal, Seborrhoeic and Acne SkinJournal of Investigative Dermatology, 1988