Nadifloxacin, an antiacne quinolone antimicrobial, inhibits the production of proinflammatory cytokines by human peripheral blood mononuclear cells and normal human keratinocytes
- 30 April 2005
- journal article
- Published by Elsevier BV in Journal of Dermatological Science
- Vol. 38 (1), 47-55
- https://doi.org/10.1016/j.jdermsci.2005.01.002
Abstract
No abstract availableKeywords
This publication has 31 references indexed in Scilit:
- Roxithromycin downmodulates Th2 chemokine production by keratinocytes and chemokine receptor expression on Th2 cells: its dual inhibitory effects on the ligands and the receptorsCellular Immunology, 2004
- Inflammatory Events Are Involved in Acne Lesion InitiationJournal of Investigative Dermatology, 2003
- Interleukin-12 and the regulation of innate resistance and adaptive immunityNature Reviews Immunology, 2003
- Concentration of Roxithromycin in the Lesions of Acne VulgarisJournal of International Medical Research, 2001
- The Macrolide Antibiotic, Roxithromycin Suppresses IFN-.GAMMA.-Mediated Immunological Functions of Cultured Normal Human Keratinocytes.Biological & Pharmaceutical Bulletin, 1996
- New understandings of the pathogenesis of acneJournal of the American Academy of Dermatology, 1995
- Cytokine Modulation of Keratinocyte CytokinesJournal of Investigative Dermatology, 1990
- Studies on antibacterial agents. I. Synthesis of substituted 6,7-dihydro-1-oxo-1H,5H-benzo(i,j)-quinolizine-2-carboxylic acids.CHEMICAL & PHARMACEUTICAL BULLETIN, 1989
- A histological and immunocytochemical study of early acne lesionsBritish Journal of Dermatology, 1988
- Delayed skin test reactivity to Propionibacterium acnes correlates with severity of inflammation in acne vulgarisBritish Journal of Dermatology, 1980