Icariin inhibits TNF-α/IFN-γ induced inflammatory response via inhibition of the substance P and p38-MAPK signaling pathway in human keratinocytes
- 1 December 2015
- journal article
- Published by Elsevier BV in International Immunopharmacology
- Vol. 29 (2), 401-407
- https://doi.org/10.1016/j.intimp.2015.10.023
Abstract
No abstract availableKeywords
Funding Information
- Development Project of Shanghai Peak Disciplines-Integrated Chinese and Western Medicine
- National Natural Science Foundation of China (81503483/H2704)
- Specialized Research Fund for the Doctoral Program of Higher Education (20120071120076)
- Shandong Provincial Medical and Health Technology Development Plan (2011HW057)
- Shanghai Science and Technology Development Funds (15401901400)
This publication has 28 references indexed in Scilit:
- A central role for spinal dorsal horn neurons that express neurokinin-1 receptors in chronic itchPain, 2015
- Effects of Dictamnus dasycarpus Turcz., root bark on ICAM-1 expression and chemokine productions in vivo and vitro studyJournal of Ethnopharmacology, 2015
- Anti-inflammatory effects of tectroside on UVB-induced HaCaT cellsInternational Journal of Molecular Medicine, 2013
- Illicium verum extract inhibits TNF-α- and IFN-γ-induced expression of chemokines and cytokines in human keratinocytesJournal of Ethnopharmacology, 2012
- Keratinocyte expression of inflammatory mediators plays a crucial role in substance P-induced acute and chronic painJournal of Neuroinflammation, 2012
- Neuroimmune interactions in allergic skin diseasesCurrent Opinion in Allergy and Clinical Immunology, 2007
- Effect of matrine on the expression of substance P receptor and inflammatory cytokines production in human skin keratinocytes and fibroblastsInternational Immunopharmacology, 2007
- Keratinocytes as initiators of inflammationThe Lancet, 1991
- Cytokine Modulation of Keratinocyte CytokinesJournal of Investigative Dermatology, 1990
- Adhesion of T lymphoblasts to epidermal keratinocytes is regulated by interferon gamma and is mediated by intercellular adhesion molecule 1 (ICAM-1).The Journal of Experimental Medicine, 1988