Ubiquilin Modifies TDP-43 Toxicity in a Drosophila Model of Amyotrophic Lateral Sclerosis (ALS)*
Open Access
- 9 April 2010
- journal article
- Published by Elsevier BV in Online Journal of Public Health Informatics
- Vol. 285 (15), 11068-11072
- https://doi.org/10.1074/jbc.c109.078527
Abstract
No abstract availableThis publication has 26 references indexed in Scilit:
- Knockdown of transactive response DNA-binding protein (TDP-43) downregulates histone deacetylase 6The EMBO Journal, 2009
- TDP-43 mutant transgenic mice develop features of ALS and frontotemporal lobar degenerationProceedings of the National Academy of Sciences of the United States of America, 2009
- Potentiation of Amyotrophic Lateral Sclerosis (ALS)-associated TDP-43 Aggregation by the Proteasome-targeting Factor, Ubiquilin 1Journal of Biological Chemistry, 2009
- Autophagy Inhibition Compromises Degradation of Ubiquitin-Proteasome Pathway SubstratesMolecular Cell, 2009
- PLIC proteins or ubiquilins regulate autophagy‐dependent cell survival during nutrient starvationEMBO Reports, 2009
- Phosphorylated TDP‐43 in frontotemporal lobar degeneration and amyotrophic lateral sclerosisAnnals of Neurology, 2008
- TDP-43 is a culprit in human neurodegeneration, and not just an innocent bystanderMammalian Genome, 2008
- Mutations inString/CDC25inhibit cell cycle re-entry and neurodegeneration in aDrosophilamodel of Ataxia telangiectasiaGenes & Development, 2008
- TDP-43 regulates retinoblastoma protein phosphorylation through the repression of cyclin-dependent kinase 6 expressionProceedings of the National Academy of Sciences of the United States of America, 2008
- The two faces of protein misfolding: gain- and loss-of-function in neurodegenerative diseasesThe EMBO Journal, 2008