Dense deposit disease
- 31 August 2011
- journal article
- review article
- Published by Elsevier BV in Molecular Immunology
- Vol. 48 (14), 1604-1610
- https://doi.org/10.1016/j.molimm.2011.04.005
Abstract
No abstract availableKeywords
This publication has 80 references indexed in Scilit:
- Common polymorphisms in C3, factor B, and factor H collaborate to determine systemic complement activity and disease riskProceedings of the National Academy of Sciences of the United States of America, 2011
- Disease-associated N-terminal Complement Factor H Mutations Perturb Cofactor and Decay-accelerating ActivitiesJournal of Biological Chemistry, 2011
- Structural basis for engagement by complement factor H of C3b on a self surfaceNature Structural & Molecular Biology, 2011
- Treatment with human complement factor H rapidly reverses renal complement deposition in factor H-deficient miceKidney International, 2010
- Factor H facilitates the clearance of GBM bound iC3b by controlling C3 activation in fluid phaseMolecular Immunology, 2009
- The disease-protective complement factor H allotypic variant Ile62 shows increased binding affinity for C3b and enhanced cofactor activityHuman Molecular Genetics, 2009
- Structure of complement fragment C3b–factor H and implications for host protection by complement regulatorsNature Immunology, 2009
- Glomeruli of Dense Deposit Disease contain components of the alternative and terminal complement pathwayKidney International, 2009
- Functional basis of protection against age-related macular degeneration conferred by a common polymorphism in complement factor BProceedings of the National Academy of Sciences of the United States of America, 2009
- Prevention of C5 activation ameliorates spontaneous and experimental glomerulonephritis in factor H-deficient miceProceedings of the National Academy of Sciences of the United States of America, 2006