Glomeruli of Dense Deposit Disease contain components of the alternative and terminal complement pathway
- 1 May 2009
- journal article
- Published by Elsevier BV in Kidney International
- Vol. 75 (9), 952-960
- https://doi.org/10.1038/ki.2008.657
Abstract
No abstract availableThis publication has 42 references indexed in Scilit:
- Factor I is required for the development of membranoproliferative glomerulonephritis in factor H–deficient miceJCI Insight, 2008
- MPGN II – genetically determined by defective complement regulation?Pediatric Nephrology, 2007
- Presence of plasma complement regulatory proteins clusterin (Apo J) and vitronectin (S40) on circulating immune complexes (CIC)Clinical and Experimental Immunology, 2006
- Prevention of C5 activation ameliorates spontaneous and experimental glomerulonephritis in factor H-deficient miceProceedings of the National Academy of Sciences of the United States of America, 2006
- Empirical Statistical Model To Estimate the Accuracy of Peptide Identifications Made by MS/MS and Database SearchAnalytical Chemistry, 2002
- Uncontrolled C3 activation causes membranoproliferative glomerulonephritis in mice deficient in complement factor HNature Genetics, 2002
- The membrane attack complex of complement induces caspase activation and apoptosisEuropean Journal of Immunology, 2002
- European Lipoprotein Club: Report of the 20th Annual Conference, Tutzing, 8–11 September 1997Atherosclerosis, 1998
- Increased frequency of apolipoprotein ε2 allele in non‐insulin dependent diabetic (NIDDM) patients with nephropathyClinical Genetics, 1995
- Incorporation of SP-40,40 into the solyble membrance attack complex(SMAC, SC5b-9) of complementInternational Immunology, 1990