Doxorubicin Induces Apoptosis and CD95 Gene Expression in Human Primary Endothelial Cells through a p53-dependent Mechanism
Open Access
- 1 March 2002
- journal article
- Published by Elsevier BV
- Vol. 277 (13), 10883-10892
- https://doi.org/10.1074/jbc.m107442200
Abstract
No abstract availableThis publication has 60 references indexed in Scilit:
- Rho GTPases Are Involved in the Regulation of NF-κB by Genotoxic StressExperimental Cell Research, 2001
- Formation of nuclear Bax/p53 complexes is associated with chemotherapy induced apoptosisOncogene, 2000
- p53-mediated up-regulation of CD95 is not involved in genotoxic drug-induced apoptosis of human breast tumor cellsCell Death & Differentiation, 1999
- BCL-2 FAMILY: Regulators of Cell DeathAnnual Review of Immunology, 1998
- The Release of Cytochrome c from Mitochondria: A Primary Site for Bcl-2 Regulation of ApoptosisScience, 1997
- FLICE, A Novel FADD-Homologous ICE/CED-3–like Protease, Is Recruited to the CD95 (Fas/APO-1) Death-Inducing Signaling ComplexCell, 1996
- Molecular cloning and expression of the fas ligand, a novel member of the tumor necrosis factor familyCell, 1993
- BCL-2 HETERODIMERIZES IN-VIVO WITH A CONSERVED HOMOLOG, BAX, THAT ACCELERATES PROGRAMMED CELL-DEATHCell, 1993
- Hepatotoxicity of mitoxantrone and doxorubicinToxicology, 1990
- Doxorubicin-induced inhibition of prolyl hydroxylation during collagen biosynthesis in human skin fibroblast cultures. Relevance to imparied wound healing.JCI Insight, 1987