SARS-CoV-2 and Guillain-Barré syndrome: molecular mimicry with human heat shock proteins as potential pathogenic mechanism
Open Access
- 29 July 2020
- journal article
- research article
- Published by Elsevier BV in Cell Stress and Chaperones
- Vol. 25 (5), 731-735
- https://doi.org/10.1007/s12192-020-01145-6
Abstract
No abstract availableKeywords
Funding Information
- Universitätsmedizin Greifswald
- Deutsche Forschungsgemeinschaft (327654276 – SFB 1315)
This publication has 30 references indexed in Scilit:
- Anti-Yo Antibody Uptake and Interaction with Its Intracellular Target Antigen Causes Purkinje Cell Death in Rat Cerebellar Slice Cultures: A Possible Mechanism for Paraneoplastic Cerebellar Degeneration in Humans with Gynecological or Breast CancersPLOS ONE, 2015
- Pathogenesis of immune-mediated neuropathiesBiochimica et Biophysica Acta (BBA) - Molecular Basis of Disease, 2015
- The immune epitope database (IEDB) 3.0Nucleic Acids Research, 2014
- Moesin is a possible target molecule for cytomegalovirus-related Guillain-Barré syndromeNeurology, 2014
- Heat-Shock Proteins in AutoimmunityAutoimmune Diseases, 2013
- Heat-Shock Proteins in Clinical NeurologyEuropean Neurology, 2011
- Autoantibodies to intracellular antigens: Generation and pathogenetic roleAutoimmunity Reviews, 2011
- UniProt Knowledgebase: a hub of integrated protein dataDatabase: The Journal of Biological Databases and Curation, 2011
- Prevalence of anti-heat shock protein antibodies in cerebrospinal fluids of patients with Guillain–Barré syndromeJournal of Neuroimmunology, 2004
- “2-6-11” motif in heat shock protein 60 and central nervous system antigens: a preliminary study in multiple sclerosis patientsJournal of Physiology and Biochemistry, 2003