The CLEC-2–podoplanin axis controls the contractility of fibroblastic reticular cells and lymph node microarchitecture
- 27 October 2014
- journal article
- research article
- Published by Springer Science and Business Media LLC in Nature Immunology
- Vol. 16 (1), 75-84
- https://doi.org/10.1038/ni.3035
Abstract
No abstract availableKeywords
This publication has 51 references indexed in Scilit:
- Podoplanin-Rich Stromal Networks Induce Dendritic Cell Motility via Activation of the C-type Lectin Receptor CLEC-2Immunity, 2012
- Transcriptional profiling of stroma from inflamed and resting lymph nodes defines immunological hallmarksNature Immunology, 2012
- Regulated release of nitric oxide by nonhematopoietic stroma controls expansion of the activated T cell pool in lymph nodesNature Immunology, 2011
- CD4 + T cells support cytotoxic T lymphocyte priming by controlling lymph node inputProceedings of the National Academy of Sciences of the United States of America, 2011
- Organizing the cell cortex: the role of ERM proteinsNature Reviews Molecular Cell Biology, 2010
- Cell-Cycle Control by Physiological Matrix Elasticity and In Vivo Tissue StiffeningCurrent Biology, 2009
- Galectin-8 interacts with podoplanin and modulates lymphatic endothelial cell functionsExperimental Cell Research, 2009
- Renal cells activate the platelet receptor CLEC-2 through podoplaninBiochemical Journal, 2008
- The potential role of podoplanin in tumour invasionBritish Journal of Cancer, 2006
- Stromal Cell Networks Regulate Lymphocyte Entry, Migration, and Territoriality in Lymph NodesImmunity, 2006