Mitofusin-2 Is a Major Determinant of Oxidative Stress-mediated Heart Muscle Cell Apoptosis
Open Access
- 1 August 2007
- journal article
- Published by Elsevier BV in Journal of Biological Chemistry
- Vol. 282 (32), 23354-23361
- https://doi.org/10.1074/jbc.m702657200
Abstract
No abstract availableKeywords
This publication has 44 references indexed in Scilit:
- Complementation between mouse Mfn1 and Mfn2 protects mitochondrial fusion defects caused by CMT2A disease mutationsThe Journal of cell biology, 2007
- Activated Mitofusin 2 Signals Mitochondrial Fusion, Interferes with Bax Activation, and Reduces Susceptibility to Radical Induced DepolarizationJournal of Biological Chemistry, 2005
- Fzo1, a Protein Involved in Mitochondrial Fusion, Inhibits ApoptosisJournal of Biological Chemistry, 2004
- Cytochrome C-Mediated ApoptosisAnnual Review of Biochemistry, 2004
- Spatial and temporal association of Bax with mitochondrial fission sites, Drp1, and Mfn2 during apoptosisThe Journal of cell biology, 2002
- Converging Pathways and Principles in Heart Development and DiseaseCell, 2002
- The Fas Signaling Pathway: More Than a ParadigmScience, 2002
- Proapoptotic BAX and BAK: A Requisite Gateway to Mitochondrial Dysfunction and DeathScience, 2001
- Reactive oxygen species and programmed cell deathTrends in Biochemical Sciences, 1996
- Reactive oxygen species and programmed cell deathTrends in Biochemical Sciences, 1996