Increased expression of TGF-β1 but not of its receptors contributes to human obstructive nephropathy
- 1 December 1999
- journal article
- Published by Elsevier BV in Kidney International
- Vol. 56 (6), 2137-2146
- https://doi.org/10.1046/j.1523-1755.1999.00790.x
Abstract
No abstract availableThis publication has 25 references indexed in Scilit:
- Fibronectin synthesis by human tubular epithelial cells in culture: Effects of PDGF and TGF-β on synthesis and splicingKidney International, 1998
- Transforming growth factor-β receptor types I and II are expressed in renal tubules and are increased after chronic unilateral ureteral obstructionLife Sciences, 1998
- Fibronectin synthesis in tubular epithelial cells: Up-regulation of the EDA splice variant by transforming growth factor βKidney International, 1995
- Mechanism of activation of the TGF-β receptorNature, 1994
- TGF-β1 in glomerulosclerosis and interstitial fibrosis of adriamycin nephropathyKidney International, 1994
- Cloning of a TGF$beta; type I receptor that forms a heteromeric complex with the TGF$beta; type II receptorCell, 1993
- Expression cloning of the TGF-β type II receptor, a functional transmembrane serine/threonine kinaseCell, 1992
- Mechanisms of tubulointerstitial fibrosisKidney International, 1991
- Transforming growth factor‐β regulates the splicing pattern of fibronectin messenger RNA precursorFEBS Letters, 1990
- Transforming growth factor β regulates the levels of different fibronectin isoforms in normal human cultured fibroblastsFEBS Letters, 1988