Reduced nicotinic receptor function in sympathetic ganglia is responsible for the hypothermia in the acetylcholinesterase knockout mouse
Open Access
- 26 January 2007
- journal article
- Published by Wiley in The Journal of Physiology
- Vol. 578 (3), 751-764
- https://doi.org/10.1113/jphysiol.2006.120147
Abstract
No abstract availableKeywords
This publication has 40 references indexed in Scilit:
- Synaptic remodeling at the skeletal neuromuscular junction of acetylcholinesterase knockout mice and its physiological relevanceChemico-Biological Interactions, 2005
- Premature Infants Are Less Capable of Maintaining Thermal Balance of Head and Body with Increases of Thermal Environment than with DecreasesAmerican Journal of Perinatology, 2005
- Reduced acetylcholine receptor density, morphological remodeling, and butyrylcholinesterase activity can sustain muscle function in acetylcholinesterase knockout miceMuscle & Nerve, 2004
- Desensitization of neuronal nicotinic receptorsJournal of Neurobiology, 2002
- Effects of Genetic Background on Thermoregulation and Fatty Acid-induced Uncoupling of Mitochondria in UCP1-deficient MicePublished by Elsevier BV ,2001
- Role of the Preoptic-Anterior Hypothalamus in Thermoregulation and FeverClinical Infectious Diseases, 2000
- Mitochondrial Uncoupling Proteins in Energy ExpenditureAnnual Review of Nutrition, 2000
- The Bioenergetics of Brown Fat Mitochondria from UCP1-ablated MiceJournal of Biological Chemistry, 1999
- Muscarinic M1receptors activate phosphoinositide turnover and Ca2+mobilisation in rat sympathetic neurones, but this signalling pathway does not mediate M‐current inhibitionThe Journal of Physiology, 1999
- Pharmacokinetics of muscarinic cholinergic drugs as determined by ex vivo (3H)-oxotremorine-M bindingJournal of Pharmacological Methods, 1990