Monocytes/macrophages prevent healing defects and left ventricular thrombus formation after myocardial infarction

Abstract
Myocardial infarction (MI) leads to rapid necrosis of cardiac myocytes. To achieve tissue integrity and function, inflammatory cells are activated, including monocytes/macrophages. However, the effec...
Funding Information
  • Deutsche Forschungsgemeinschaft (SFB688 TP A10)