Macrophage Depletion Impairs Wound Healing and Increases Left Ventricular Remodeling after Myocardial Injury in Mice
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Open Access
- 1 March 2007
- journal article
- Published by Elsevier BV in The American Journal of Pathology
- Vol. 170 (3), 818-829
- https://doi.org/10.2353/ajpath.2007.060547
Abstract
No abstract availableKeywords
This publication has 35 references indexed in Scilit:
- Ex Vivo Activated Human Macrophages Improve Healing, Remodeling, and Function of the Infarcted HeartCirculation, 2006
- Macrophage Colony-Stimulating Factor Treatment After Myocardial Infarction Attenuates Left Ventricular Dysfunction by Accelerating Infarct RepairJournal of the American College of Cardiology, 2006
- Tenascin-C Regulates Recruitment of Myofibroblasts during Tissue Repair after Myocardial InjuryThe American Journal of Pathology, 2005
- Therapeutic angiogenesis in chronically ischemic porcine myocardium: comparative effects of bFGF and VEGFThe Annals of Thoracic Surgery, 2004
- Anti-Monocyte Chemoattractant Protein-1 Gene Therapy Attenuates Left Ventricular Remodeling and Failure After Experimental Myocardial InfarctionCirculation, 2003
- Macrophage Depletion by Clodronate-Containing Liposomes Reduces Neointimal Formation After Balloon Injury in Rats and RabbitsCirculation, 2002
- Vascular Endothelial Growth Factor, a Potent and Selective Angiogenic AgentJournal of Biological Chemistry, 1996
- Transforming growth factor-beta 1 induces alpha-smooth muscle actin expression in granulation tissue myofibroblasts and in quiescent and growing cultured fibroblastsThe Journal of cell biology, 1993
- Cardiac Cryolesions as an Experimental Model of Myocardial Wound HealingAnnals of Surgery, 1987
- Accelerated Healing of Incisional Wounds in Rats Induced by Transforming Growth Factor-βScience, 1987