The IκB Kinase Regulates Chromatin Structure during Reconsolidation of Conditioned Fear Memories
- 20 September 2007
- journal article
- research article
- Published by Elsevier BV in Neuron
- Vol. 55 (6), 942-957
- https://doi.org/10.1016/j.neuron.2007.07.039
Abstract
No abstract availableKeywords
Funding Information
- Epilepsy Foundation
- National Institutes of Health (MH57014, NS048811, NS37444)
- American Health Assistance Foundation
This publication has 74 references indexed in Scilit:
- A transcription factor-binding domain of the coactivator CBP is essential for long-term memory and the expression of specific target genesLearning & Memory, 2006
- ERK/MAPK regulates hippocampal histone phosphorylation following contextual fear conditioningLearning & Memory, 2006
- Characterization of Fear Memory ReconsolidationJournal of Neuroscience, 2004
- IκB Kinase α and p65/RelA Contribute to Optimal Epidermal Growth Factor-induced c-fos Gene Expression Independent of IκBα DegradationPublished by Elsevier BV ,2004
- Independent Cellular Processes for Hippocampal Memory Consolidation and ReconsolidationScience, 2004
- A Bioinformatics Analysis of Memory Consolidation Reveals Involvement of the Transcription Factor c-RelJournal of Neuroscience, 2004
- Acetylation of Nuclear Factor-κB in Rat Amygdala Improves Long-Term but not Short-Term Retention of Fear MemoryMolecular Pharmacology, 2004
- Post-activation Turn-off of NF-κB-dependent Transcription Is Regulated by Acetylation of p65Journal of Biological Chemistry, 2003
- Positive and Negative Regulation of IκB Kinase Activity Through IKKβ Subunit PhosphorylationScience, 1999
- THE NF-κB AND IκB PROTEINS: New Discoveries and InsightsAnnual Review of Immunology, 1996