Post-activation Turn-off of NF-κB-dependent Transcription Is Regulated by Acetylation of p65
Open Access
- 1 January 2003
- journal article
- Published by Elsevier BV in Journal of Biological Chemistry
- Vol. 278 (4), 2758-2766
- https://doi.org/10.1074/jbc.m209572200
Abstract
No abstract availableKeywords
This publication has 41 references indexed in Scilit:
- Cell-Specific Association and Shuttling of IκBα Provides a Mechanism for Nuclear NF-κB in B LymphocytesMolecular and Cellular Biology, 2001
- Cyclin A Is a Mediator of p120E4F-Dependent Cell Cycle Arrest in G1Molecular and Cellular Biology, 2001
- Mechanism of IκBα Binding to NF-κB DimersOnline Journal of Public Health Informatics, 2000
- How NF-κB is activated: the role of the IκB kinase (IKK) complexOncogene, 1999
- Activators and target genes of Rel/NF-κB transcription factorsOncogene, 1999
- Regulation of DNA binding by Rel/NF-κB transcription factors: structural viewsOncogene, 1999
- HIV-1 Tat transcriptional activity is regulated by acetylationThe EMBO Journal, 1999
- Phosphorylation of NF-κB p65 by PKA Stimulates Transcriptional Activity by Promoting a Novel Bivalent Interaction with the Coactivator CBP/p300Molecular Cell, 1998
- NF-κB AND REL PROTEINS: Evolutionarily Conserved Mediators of Immune ResponsesAnnual Review of Immunology, 1998
- Analysis of Chromatin Structure byin VivoFormaldehyde Cross-LinkingMethods, 1997