Acute δ-opioid receptor activation induces CREB phosphorylation in NG108-15 cells
- 1 February 2000
- journal article
- Published by Elsevier BV in European Journal of Pharmacology
- Vol. 390 (1-2), 1-6
- https://doi.org/10.1016/s0014-2999(00)00018-2
Abstract
No abstract availableKeywords
This publication has 31 references indexed in Scilit:
- Desensitization and resensitization of δ-opioid receptor-mediated Ca2+channel inhibition in NG108-15 cellsBritish Journal of Pharmacology, 1998
- δ‐Opioids Stimulate Inositol 1,4,5‐Trisphosphate Formation, and so Mobilize Ca2+ from Intracellular Stores, in Undifferentiated NG108‐15 CellsJournal of Neurochemistry, 1996
- Transcriptional Regulation of CREB (Cyclic AMP Response Element‐Binding Protein) Expression in CATH.a CellsJournal of Neurochemistry, 1996
- Decrease in CRE binding activity by chronic morphine administration in mouse brainNeuroReport, 1995
- Influence of chronic morphine treatment on protein kinase C activity: comparison with butorphanol and implication for opioid toleranceBrain Research, 1994
- Drug addiction: A model for the molecular basis of neural plasticityNeuron, 1993
- Regulation of CREB Phosphorylation in the Suprachiasmatic Nucleus by Light and a Circadian ClockScience, 1993
- CREB: a Ca 2+ -Regulated Transcription Factor Phosphorylated by Calmodulin-Dependent KinasesScience, 1991
- Membrane depolarization and calcium induce c-fos transcription via phosphorylation of transcription factor CREBNeuron, 1990
- Phosphorylation-induced binding and transcriptional efficacy of nuclear factor CREBNature, 1988