Intestinal Deletion of Pofut1 in the Mouse Inactivates Notch Signaling and Causes Enterocolitis
Open Access
- 30 September 2008
- journal article
- Published by Elsevier BV in Gastroenterology
- Vol. 135 (3), 849-860.e6
- https://doi.org/10.1053/j.gastro.2008.05.050
Abstract
No abstract availableThis publication has 36 references indexed in Scilit:
- Roles of Pofut1 and O-Fucose in Mammalian Notch SignalingJournal of Biological Chemistry, 2008
- Loss of intestinal crypt progenitor cells owing to inactivation of both Notch1 and Notch2 is accompanied by derepression of CDK inhibitors p27Kip1 and p57Kip2EMBO Reports, 2008
- Sox9 regulates cell proliferation and is required for Paneth cell differentiation in the intestinal epitheliumThe Journal of cell biology, 2007
- Comparative study of the intestinal mucus barrier in normal and inflamed colonGut, 2007
- Mapping the consequence of Notch1 proteolysis in vivo with NIP-CREDevelopment, 2007
- The roles of mucus-associated bacteria in inflammatory bowel diseaseDrugs of Today, 2006
- Bacterial Overgrowth in the Cystic Fibrosis Transmembrane Conductance Regulator Null Mouse Small IntestineInfection and Immunity, 2004
- AnO-fucose site in the ligand binding domain inhibits Notch activationDevelopment, 2003
- Functional Diversity among Notch1, Notch2, and Notch3 ReceptorsBiochemical and Biophysical Research Communications, 2002
- Signalling downstream of activated mammalian NotchNature, 1995