Fibrosis Rescue Improves Cardiac Function in Dystrophin-Deficient Mice and Duchenne Patient–Specific Cardiomyocytes by Immunoproteasome Modulation
- 15 November 2018
- journal article
- research article
- Published by Elsevier BV in The American Journal of Pathology
- Vol. 189 (2), 339-353
- https://doi.org/10.1016/j.ajpath.2018.10.010
Abstract
No abstract availableKeywords
Funding Information
- Ministry of Health
- Fondazione Umberto Veronesi
- Fondazione IEO CCM
This publication has 51 references indexed in Scilit:
- Dystrophin-Deficient CardiomyopathyJournal of the American College of Cardiology, 2016
- Pathophysiology and Therapy of Cardiac Dysfunction in Duchenne Muscular DystrophyAmerican Journal of Cardiovascular Drugs, 2011
- Cardiac Involvement in Patients With Muscular DystrophiesCirculation: Cardiovascular Imaging, 2011
- Cardiac involvement in muscular dystrophiesQJM: An International Journal of Medicine, 2008
- Role of the JAK–STAT pathway in myocardial injuryTrends in Molecular Medicine, 2007
- Intracellular calcium handling in ventricular myocytes from mdx miceAmerican Journal of Physiology-Heart and Circulatory Physiology, 2007
- Evolution of the mdx mouse cardiomyopathy: physiological and morphological findingsNeuromuscular Disorders, 2004
- Progression of dystrophic features and activation of mitogen‐activated protein kinases and calcineurin by physical exercise, in hearts of mdx miceFEBS Letters, 2002
- An essential role of the JAK-STAT pathway in ischemic preconditioningProceedings of the National Academy of Sciences of the United States of America, 2001
- Dystrophin‐deficient cardiomyocytes are abnormally vulnerable to mechanical stress‐induced contractile failure and injuryThe FASEB Journal, 2001