Dystrophin‐deficient cardiomyocytes are abnormally vulnerable to mechanical stress‐induced contractile failure and injury
- 29 May 2001
- journal article
- Published by Wiley in The FASEB Journal
- Vol. 15 (9), 1655-1657
- https://doi.org/10.1096/fj.01-0030fje
Abstract
No abstract availableFunding Information
- Hospital for Sick Children
- Canadian Institutes of Health Research
- Fonds de Recherche du Québec - Santé
- Muscular Dystrophy Association
This publication has 38 references indexed in Scilit:
- Disruption of the Sarcoglycan–Sarcospan Complex in Vascular Smooth MuscleCell, 1999
- Evidence ofmdx mouse skeletal muscle fragility in vivo by eccentric running exerciseMuscle & Nerve, 1998
- Dystrobrevin and dystrophin: An interaction through coiled-coil motifsProceedings of the National Academy of Sciences of the United States of America, 1997
- Animal Models for Muscular Dystrophy Show Different Patterns of Sarcolemmal DisruptionThe Journal of cell biology, 1997
- Dystrophies and heart diseaseCurrent Opinion in Cardiology, 1997
- A role for the dystrophin-glycoprotein complex as a transmembrane linker between laminin and actinThe Journal of cell biology, 1993
- Dystrophin protects the sarcolemma from stresses developed during muscle contraction.Proceedings of the National Academy of Sciences of the United States of America, 1993
- Dystrophin-deficient mdx muscle fibers are preferentially vulnerable to necrosis induced by experimental lengthening contractionsJournal of the Neurological Sciences, 1990
- The Molecular Basis of Muscular Dystrophy in the mdx Mouse: a Point MutationScience, 1989
- Dystrophin: The protein product of the duchenne muscular dystrophy locusCell, 1987