Fatty Acid Oxidation Mediated by Acyl-CoA Synthetase Long Chain 3 Is Required for Mutant KRAS Lung Tumorigenesis
Open Access
- 28 July 2016
- journal article
- research article
- Published by Elsevier BV in Cell Reports
- Vol. 16 (6), 1614-1628
- https://doi.org/10.1016/j.celrep.2016.07.009
Abstract
No abstract availableThis publication has 51 references indexed in Scilit:
- Hypoxic and Ras-transformed cells support growth by scavenging unsaturated fatty acids from lysophospholipidsProceedings of the National Academy of Sciences of the United States of America, 2013
- mTOR Signaling in Growth Control and DiseaseCell, 2012
- Oncogenic Kras Maintains Pancreatic Tumors through Regulation of Anabolic Glucose MetabolismCell, 2012
- Evidence for type II cells as cells of origin of K-Ras–induced distal lung adenocarcinomaProceedings of the National Academy of Sciences of the United States of America, 2012
- RAS oncogenes: weaving a tumorigenic webNature Reviews Cancer, 2011
- K-rasG12V transformation leads to mitochondrial dysfunction and a metabolic switch from oxidative phosphorylation to glycolysisCell Research, 2011
- Hallmarks of Cancer: The Next GenerationCell, 2011
- Conditional mouse lung cancer models using adenoviral or lentiviral delivery of Cre recombinaseNature Protocols, 2009
- A Gene Expression Signature Associated with “K-Ras Addiction” Reveals Regulators of EMT and Tumor Cell SurvivalCancer Cell, 2009
- Significance analysis of microarrays applied to the ionizing radiation responseProceedings of the National Academy of Sciences of the United States of America, 2001