Pathogenic Role of IL-6 Combined with TNF-α or IL-1 in the Induction of Acute Phase Proteins SAA and CRP in Chronic Inflammatory Diseases
- 22 October 2010
- book chapter
- Published by Springer Science and Business Media LLC in Advances in experimental medicine and biology
- Vol. 691, 141-150
- https://doi.org/10.1007/978-1-4419-6612-4_15
Abstract
No abstract availableKeywords
This publication has 15 references indexed in Scilit:
- Transcriptional Complex Formation of c-Fos, STAT3, and Hepatocyte NF-1α Is Essential for Cytokine-Driven C-Reactive Protein Gene ExpressionPublished by The American Association of Immunologists ,2008
- Humanized anti–interleukin-6 receptor antibody treatment of multicentric Castleman diseaseBlood, 2005
- Essential role of STAT3 in cytokine‐driven NF‐κB‐mediated serum amyloid A gene expressionGenes to Cells, 2005
- Therapeutic efficacy of humanized recombinant anti–interleukin‐6 receptor antibody in children with systemic‐onset juvenile idiopathic arthritisArthritis & Rheumatism, 2005
- Treatment of rheumatoid arthritis with humanized anti–interleukin‐6 receptor antibody: A multicenter, double‐blind, placebo‐controlled trialArthritis & Rheumatism, 2004
- IL-6 plays a critical role in the synergistic induction of human serum amyloid A (SAA) gene when stimulated with proinflammatory cytokines as analyzed with an SAA isoform real-time quantitative RT-PCR assay systemBiochemical and Biophysical Research Communications, 2004
- Interleukin-6-Induced STAT3 and AP-1 Amplify Hepatocyte Nuclear Factor 1-Mediated Transactivation of Hepatic Genes, an Adaptive Response to Liver InjuryMolecular and Cellular Biology, 2001
- Interacting Regions in Stat3 and c-Jun That Participate in Cooperative Transcriptional ActivationMolecular and Cellular Biology, 1999
- Synergistic Signaling in Fetal Brain by STAT3-Smad1 Complex Bridged by p300Science, 1999
- STAT3 Participates in Transcriptional Activation of the C-reactive Protein Gene by Interleukin-6Published by Elsevier BV ,1996