Preferential neuroprotective effect of tacrolimus (FK506) on unmyelinated axons following traumatic brain injury
- 18 June 2007
- journal article
- Published by Elsevier BV in Brain Research
- Vol. 1154, 225-236
- https://doi.org/10.1016/j.brainres.2007.04.002
Abstract
No abstract availableKeywords
This publication has 48 references indexed in Scilit:
- Administration of the immunophilin ligand FK506 differentially attenuates neurofilament compaction and impaired axonal transport in injured axons following diffuse traumatic brain injuryExperimental Neurology, 2006
- FK506 Treatment Inhibits Caspase-3 Activation and Promotes Oligodendroglial Survival Following Traumatic Spinal Cord InjuryExperimental Neurology, 2002
- Attenuation of the Electrophysiological Function of the Corpus Callosum after Fluid Percussion Injury in the RatJournal of Neurotrauma, 2002
- The Immunophilin Ligand FK506 Attenuates the Axonal Damage Associated with Rapid Rewarming Following Posttraumatic HypothermiaExperimental Neurology, 2001
- Effects of tacrolimus on hemispheric water content and cerebrospinal fluid levels of glutamate, hypoxanthine, interleukin-6, and tumor necrosis factor-α following controlled cortical impact injury in ratsJournal of Neurosurgery, 2001
- Axonal Damage in Traumatic Brain InjuryThe Neuroscientist, 2000
- Tacrolimus (FK506) Increases Neuronal Expression of GAP-43 and Improves Functional Recovery after Spinal Cord Injury in RatsExperimental Neurology, 1998
- Maturation of the corpus callosum of the rat: II. Influence of thyroid hormones on the number and maturation of axonsJournal of Comparative Neurology, 1990
- Dephosphorylation of Microtubule‐Associated Protein 2, τ Factor, and Tubulin by CalcineurinJournal of Neurochemistry, 1985
- MYELINATION OF THE MOUSE CORPUS CALLOSUMNeuropathology and Applied Neurobiology, 1980