Tacrolimus (FK506) Increases Neuronal Expression of GAP-43 and Improves Functional Recovery after Spinal Cord Injury in Rats
- 1 December 1998
- journal article
- Published by Elsevier BV in Experimental Neurology
- Vol. 154 (2), 673-683
- https://doi.org/10.1006/exnr.1998.6974
Abstract
No abstract availableKeywords
This publication has 43 references indexed in Scilit:
- Molecular Biology of Axonal OutgrowthPediatric Neurosurgery, 1997
- Suppression of Experimental Crescentic-Type Anti-Glomerular Basement Membrane (GBM) Nephritis by FK506 (Tacrolimus Hydrate) in RatsThe Japanese Journal of Pharmacology, 1996
- Design, synthesis and structure of non-macrocyclic inhibitors of FKBP12, the major binding protein for the immunosuppressant FK506Acta Crystallographica Section D-Biological Crystallography, 1995
- The neuroprotective effect of high-dose methylprednisolone in rat spinal cord hemisectionNeuroscience Letters, 1995
- The relationships among the severity of spinal cord injury, residual neurological function, axon counts, and counts of retrogradely labeled neurons after experimental spinal cord injuryExperimental Neurology, 1995
- Absence of persistent spreading, branching, and adhesion in GAP-43-depleted growth cones.The Journal of cell biology, 1995
- A Sensitive and Reliable Locomotor Rating Scale for Open Field Testing in RatsJournal of Neurotrauma, 1995
- Up-regulation of GAP-43 and growth of axons in rat spinal cord after compression injuryJournal of Neurocytology, 1993
- Protective Effect of Methylprednisolone on Vascular Injury in Rat Spinal Cord InjuryJournal of Neurotrauma, 1992
- Monoclonal antibodies show that kinase C phosphorylation of GAP-43 during axonogenesis is both spatially and temporally restricted in vivo.The Journal of cell biology, 1991