Oligodendrocyte Apoptosis and Primary Demyelination Induced by Local TNF/p55TNF Receptor Signaling in the Central Nervous System of Transgenic Mice: Models for Multiple Sclerosis with Primary Oligodendrogliopathy
- 30 September 1998
- journal article
- Published by Elsevier BV in The American Journal of Pathology
- Vol. 153 (3), 801-813
- https://doi.org/10.1016/s0002-9440(10)65622-2
Abstract
No abstract availableKeywords
Funding Information
- European Commission (1629, BIO4-CT96–0174, BIO4-CT96–0077, ASTF 8543)
This publication has 72 references indexed in Scilit:
- Late Complications of Immune Deviation Therapy in a Nonhuman PrimateScience, 1996
- Dissection of TNF Receptor 1 Effector Functions: JNK Activation Is Not Linked to Apoptosis While NF-κB Activation Prevents Cell DeathCell, 1996
- Cytokines in inflammatory brain lesions: helpful and harmfulTrends in Neurosciences, 1996
- Mechanisms of Immune Injury in Multiple SclerosisBrain Pathology, 1996
- Two models of multiple sclerosis: Experimental allergic encephalomyelitis (EAE) and theiler's murine encephalomyelitis virus (TMEV) infection. A pathological and immunological comparisonMicroscopy Research and Technique, 1995
- Tumor necrosis factors protect neurons against metabolic-excitotoxic insults and promote maintenance of calcium homeostasisNeuron, 1994
- Animal modelsAnnals of Neurology, 1994
- The Pathophysiology of Tumor Necrosis FactorsAnnual Review of Immunology, 1992
- Immunological Aspects of Demyelinating DiseasesAnnual Review of Immunology, 1992
- The T Lymphocyte in Experimental Allergic EncephalomyelitisAnnual Review of Immunology, 1990