Rip1 (Receptor-interacting protein kinase 1) mediates necroptosis and contributes to renal ischemia/reperfusion injury
- 1 April 2012
- journal article
- Published by Elsevier BV in Kidney International
- Vol. 81 (8), 751-761
- https://doi.org/10.1038/ki.2011.450
Abstract
No abstract availableKeywords
This publication has 53 references indexed in Scilit:
- cIAPs Block Ripoptosome Formation, a RIP1/Caspase-8 Containing Intracellular Cell Death Complex Differentially Regulated by cFLIP IsoformsMolecular Cell, 2011
- RIP3 mediates the embryonic lethality of caspase-8-deficient miceNature, 2011
- Catalytic activity of the caspase-8–FLIPL complex inhibits RIPK3-dependent necrosisNature, 2011
- Multiple death pathways in TNF-treated fibroblasts: RIP3- and RIP1-dependent and independent routesCell Research, 2011
- Necrostatin Decreases Oxidative Damage, Inflammation, and Injury after Neonatal HIJournal of Cerebral Blood Flow & Metabolism, 2010
- Going up in flames: necrotic cell injury and inflammatory diseasesCellular and Molecular Life Sciences, 2010
- Necroptosis as an alternative form of programmed cell deathCurrent Opinion in Cell Biology, 2010
- Phosphorylation-Driven Assembly of the RIP1-RIP3 Complex Regulates Programmed Necrosis and Virus-Induced InflammationCell, 2009
- Receptor Interacting Protein Kinase-3 Determines Cellular Necrotic Response to TNF-αCell, 2009
- Guidelines for the use and interpretation of assays for monitoring cell death in higher eukaryotesCell Death & Differentiation, 2009