Eyes absent 1 (Eya1) is a critical coordinator of epithelial, mesenchymal and vascular morphogenesis in the mammalian lung
- 1 February 2011
- journal article
- Published by Elsevier BV in Developmental Biology
- Vol. 350 (1), 112-126
- https://doi.org/10.1016/j.ydbio.2010.11.022
Abstract
No abstract availableKeywords
Funding Information
- NIH-NHLBI (P01 HL 60231, HL 44060, HL44977)
- California Institute of Regenerative Medicine (CIRM)
This publication has 89 references indexed in Scilit:
- Conditional gene inactivation reveals roles for Fgf10 and Fgfr2 in establishing a normal pattern of epithelial branching in the mouse lungDevelopmental Dynamics, 2009
- miR-17 family of microRNAs controls FGF10-mediated embryonic lung epithelial branching morphogenesis through MAPK14 and STAT3 regulation of E-Cadherin distributionDevelopmental Biology, 2009
- Interactions with the Abelson Tyrosine Kinase Reveal Compartmentalization of Eyes Absent Function between Nucleus and CytoplasmDevelopmental Cell, 2009
- Eya1 gene dosage critically affects the development of sensory epithelia in the mammalian inner earHuman Molecular Genetics, 2008
- Order in the lungNature, 2008
- Eya1 and Six1 promote neurogenesis in the cranial placodes in a SoxB1-dependent fashionDevelopmental Biology, 2008
- The branching programme of mouse lung developmentNature, 2008
- Sonic hedgehog is an autocrine viability factor for myofibroblastic hepatic stellate cellsJournal of Hepatology, 2007
- Eya protein phosphatase activity regulates Six1–Dach–Eya transcriptional effects in mammalian organogenesisNature, 2003
- A human homologue of the Drosophila eyes absent gene underlies Branchio-Oto-Renal (BOR) syndrome and identifies a novel gene familyNature Genetics, 1997