SK2 Channels Are Neuroprotective for Ischemia-Induced Neuronal Cell Death
- 29 June 2011
- journal article
- Published by SAGE Publications in Journal of Cerebral Blood Flow & Metabolism
- Vol. 31 (12), 2302-2312
- https://doi.org/10.1038/jcbfm.2011.90
Abstract
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This publication has 41 references indexed in Scilit:
- Ischemic insult to cerebellar Purkinje cells causes diminished GABAA receptor function and allopregnanolone neuroprotection is associated with GABAA receptor stabilizationJournal of Neurochemistry, 2008
- Histopathological and behavioral characterization of a novel model of cardiac arrest and cardiopulmonary resuscitation in miceJournal of Neuroscience Methods, 2004
- Ischemia induces short‐ and long‐term remodeling of synaptic activity in the hippocampusJournal of Cellular and Molecular Medicine, 2003
- Mechanisms, challenges and opportunities in strokeNature Reviews Neuroscience, 2003
- Small Conductance Ca2+-Activated K+Channels Modulate Synaptic Plasticity and Memory EncodingJournal of Neuroscience, 2002
- A Novel N-Methyl-d-aspartate Receptor Open Channel Blocker with in Vivo Neuroprotectant ActivityJournal of Pharmacology and Experimental Therapeutics, 2002
- Extrasynaptic NMDARs oppose synaptic NMDARs by triggering CREB shut-off and cell death pathwaysNature Neuroscience, 2002
- Treatment of Comatose Survivors of Out-of-Hospital Cardiac Arrest with Induced HypothermiaNew England Journal of Medicine, 2002
- Therapeutic Hypothermia after Cardiac ArrestNew England Journal of Medicine, 2002
- Control of Electrical Activity in Central Neurons by Modulating the Gating of Small Conductance Ca2+-activated K+ ChannelsPublished by Elsevier BV ,2001