Cross-talk between the p38α and JNK MAPK Pathways Mediated by MAP Kinase Phosphatase-1 Determines Cellular Sensitivity to UV Radiation
Open Access
- 1 August 2010
- journal article
- Published by Elsevier BV in Online Journal of Public Health Informatics
- Vol. 285 (34), 25928-25940
- https://doi.org/10.1074/jbc.m110.117911
Abstract
No abstract availableThis publication has 64 references indexed in Scilit:
- cAMP-response Element-binding Protein (CREB) Controls MSK1-mediated Phosphorylation of Histone H3 at the c-fos Promoter in VitroOnline Journal of Public Health Informatics, 2010
- MKP-1 Is Necessary for T Cell Activation and FunctionOnline Journal of Public Health Informatics, 2009
- Signal integration by JNK and p38 MAPK pathways in cancer developmentNature Reviews Cancer, 2009
- Histone H3 as a novel substrate for MAP kinase phosphatase-1American Journal of Physiology-Cell Physiology, 2009
- The kinase p38α serves cell type–specific inflammatory functions in skin injury and coordinates pro- and anti-inflammatory gene expressionNature Immunology, 2008
- MKP-1 mRNA Stabilization and Translational Control by RNA-Binding Proteins HuR and NF90Molecular and Cellular Biology, 2008
- Mitogen-activated Protein Kinase Phosphatase-1 Represses c-Jun NH2-terminal Kinase-mediated Apoptosis via NF-κB RegulationOnline Journal of Public Health Informatics, 2008
- Negative-feedback regulation of FGF signalling by DUSP6/MKP-3 is driven by ERK1/2 and mediated by Ets factor binding to a conserved site within the DUSP6/MKP-3 gene promoterBiochemical Journal, 2008
- Feedback regulation of p38 activity via ATF2 is essential for survival of embryonic liver cellsGenes & Development, 2007
- Regulation of innate immune response by MAP kinase phosphatase-1Cellular Signalling, 2007