Impairment of an Endothelial NAD+-H2S Signaling Network Is a Reversible Cause of Vascular Aging
Top Cited Papers
- 1 March 2018
- journal article
- research article
- Published by Elsevier BV in Cell
- Vol. 173 (1), 74-89.e20
- https://doi.org/10.1016/j.cell.2018.02.008
Abstract
No abstract availableKeywords
Funding Information
- Glenn Foundation for Medical Research (RO1 AG028730, RO1 DK100263)
- Sinclair Gift Fund
- Edward Schulak (APP1093643)
- NIH/NIA (RO1 AG015339)
- NIH/NHLBI (RO1 HL094499)
This publication has 84 references indexed in Scilit:
- The NAD+ Precursor Nicotinamide Riboside Enhances Oxidative Metabolism and Protects against High-Fat Diet-Induced ObesityCell Metabolism, 2012
- SIRT1 Is Required for AMPK Activation and the Beneficial Effects of Resveratrol on Mitochondrial FunctionCell Metabolism, 2012
- Nicotinamide Mononucleotide, a Key NAD+ Intermediate, Treats the Pathophysiology of Diet- and Age-Induced Diabetes in MiceCell Metabolism, 2011
- PARP-1 Inhibition Increases Mitochondrial Metabolism through SIRT1 ActivationCell Metabolism, 2011
- PARP-2 Regulates SIRT1 Expression and Whole-Body Energy ExpenditureCell Metabolism, 2011
- AMPK regulates energy expenditure by modulating NAD+ metabolism and SIRT1 activityNature, 2009
- AMPK and PPARδ Agonists Are Exercise MimeticsCell, 2008
- Resveratrol Delays Age-Related Deterioration and Mimics Transcriptional Aspects of Dietary Restriction without Extending Life SpanCell Metabolism, 2008
- Resveratrol improves health and survival of mice on a high-calorie dietNature, 2006
- Transcriptional co-activator PGC-1α drives the formation of slow-twitch muscle fibresNature, 2002