Hemiballism-hemichorea and non-ketotic hyperglycaemia.
Open Access
- 1 June 1994
- journal article
- case report
- Published by BMJ in Journal of Neurology, Neurosurgery & Psychiatry
- Vol. 57 (6), 748-750
- https://doi.org/10.1136/jnnp.57.6.748
Abstract
Three patients with hemiballism-hemichorea caused by non-ketotic hyperglycaemia are presented, two of whom had hyperosmolar non-ketotic hyperglycaemic syndrome. In two of the three patients, the hyperkinesia was the initial presenting symptom of their diabetes mellitus. The hypersensitivity of the postmenopausal dopamine receptor, decreased gamma-aminobutyric acid in the brain in non-ketotic hyperglycaemia, coexisting lacunar infarct in the basal ganglion, and pre-existing metabolic dysfunction in the basal ganglion may all have played a part in the pathogenesis of this movement disorder.Keywords
This publication has 15 references indexed in Scilit:
- Alternating Choreoathetosis Associated with Uncontrolled Diabetes Mellitus and Basal Ganglia CalcificationDiabetes Care, 1986
- Experimental hemichorea/hemiballismus in the monkey. Studies on the intracerebral site of action in a drug-induced dyskinesia.1984
- Paroxysmal Choreoathetosis due to HypoglycemiaArchives of Neurology, 1984
- Nonketotic Hyperglycemia Appearing as Choreoathetosis or BallismArchives of Internal Medicine, 1982
- Increased Dopamine Receptor Sensitivity After Estrogen Treatment Using the Rat Rotation ModelScience, 1980
- Modulation of dopamine receptor sensitivity by estrogen.1980
- Dopamine and noradrenaline in post-mortem brain in Huntington's disease and schizophrenic illness.1980
- Treatment of vascular hemiballism and hemichoreaNeurology, 1977
- Embolic Mononeuropathy and Bacterial EndocarditisArchives of Neurology, 1968
- Some observations on hemiballismusNeurology, 1960