Optimization of the Antitumor Efficacy of a Synthetic Mitochondrial Toxin by Increasing the Residence Time in the Cytosol
- 29 September 2009
- journal article
- research article
- Published by American Chemical Society (ACS) in Journal of Medicinal Chemistry
- Vol. 52 (20), 6209-6216
- https://doi.org/10.1021/jm9008339
Abstract
No abstract availableKeywords
This publication has 17 references indexed in Scilit:
- Controlling metabolism and cell death: At the heart of mitochondrial calcium signallingJournal of Molecular and Cellular Cardiology, 2009
- What is the mitochondrial permeability transition pore?Journal of Molecular and Cellular Cardiology, 2009
- Metabolism of the Tumor Angiogenesis Inhibitor 4-(N-(S-Glutathionylacetyl)amino)phenylarsonous AcidPublished by Elsevier BV ,2008
- Mechanism of Selectivity of an Angiogenesis Inhibitor From Screening a Genome-Wide Set of Saccharomyces cerevisiae Deletion StrainsJNCI Journal of the National Cancer Institute, 2005
- Mitochondria as cancer drug targetsTrends in Molecular Medicine, 2004
- A peptide trivalent arsenical inhibits tumor angiogenesis by perturbing mitochondrial function in angiogenic endothelial cellsCancer Cell, 2003
- Involvement of Human Organic Anion Transporting Polypeptide OATP-B (SLC21A9) in pH-Dependent Transport across Intestinal Apical MembraneThe Journal of pharmacology and experimental therapeutics, 2003
- Role of critical thiol groups on the matrix surface of the adenine nucleotide translocase in the mechanism of the mitochondrial permeability transition poreBiochemical Journal, 2002
- The permeability transition pore complex: another viewBiochimie, 2002
- Chemistry of organometalloid complexes with potential antidotes: structure of an organoarsenic(III) dithiolate ringInorganic Chemistry, 1990