TGF-β activates Akt kinase through a microRNA-dependent amplifying circuit targeting PTEN
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Open Access
- 21 June 2009
- journal article
- research article
- Published by Springer Science and Business Media LLC in Nature
- Vol. 11 (7), 881-889
- https://doi.org/10.1038/ncb1897
Abstract
No abstract availableKeywords
This publication has 34 references indexed in Scilit:
- MicroRNAs: Target Recognition and Regulatory FunctionsCell, 2009
- Tyrosine phosphorylation-dependent PI 3 kinase/Akt signal transduction regulates TGFβ-induced fibronectin expression in mesangial cellsCellular Signalling, 2004
- The Phosphatidylinositol 3-Kinase/Akt Pathway Enhances Smad3-stimulated Mesangial Cell Collagen I Expression in Response to Transforming Growth Factor-β1Online Journal of Public Health Informatics, 2004
- Vertebrate MicroRNA GenesScience, 2003
- Signalling pathways that mediate skeletal muscle hypertrophy and atrophyNature, 2003
- Mutation and Expression Analyses Reveal Differential Subcellular Compartmentalization of PTEN in Endocrine Pancreatic Tumors Compared to Normal Islet CellsThe American Journal of Pathology, 2000
- Synergistic cooperation of TFE3 and Smad proteins in TGF-β-induced transcription of the plasminogen activator inhibitor-1 geneGenes & Development, 1998
- Hyperglycemia and Diabetic Kidney Disease: The Case for Transforming Growth Factor–β as a Key MediatorDiabetes, 1995
- The delta-crystallin enhancer-binding protein delta EF1 is a repressor of E2-box-mediated gene activation.Molecular and Cellular Biology, 1994
- Expression of transforming growth factor beta is elevated in human and experimental diabetic nephropathy.Proceedings of the National Academy of Sciences of the United States of America, 1993