Metalloproteinase-mediated Shedding of Integrin β2 Promotes Macrophage Efflux from Inflammatory Sites
Open Access
- 1 February 2012
- journal article
- Published by Elsevier BV in Online Journal of Public Health Informatics
- Vol. 287 (7), 4581-4589
- https://doi.org/10.1074/jbc.m111.321182
Abstract
No abstract availableThis publication has 38 references indexed in Scilit:
- Molecular Basis for the Interaction of Low Density Lipoprotein Receptor-related Protein 1 (LRP1) with Integrin αMβ2Online Journal of Public Health Informatics, 2011
- Adam17-dependent shedding limits early neutrophil influx but does not alter early monocyte recruitment to inflammatory sitesBlood, 2011
- Cleavage of the CD11b extracellular domain by the leukocyte serprocidins is critical for neutrophil detachment during chemotaxisBlood, 2011
- Saturated‐efferocytosis generates pro‐resolving CD11blow macrophages: Modulation by resolvins and glucocorticoidsEuropean Journal of Immunology, 2010
- ADAM10 is essential for Notch2-dependent marginal zone B cell development and CD23 cleavage in vivoThe Journal of Experimental Medicine, 2010
- MMP-9 Sheds the β2 Integrin Subunit (CD18) from MacrophagesMolecular & Cellular Proteomics, 2009
- Inflammatory macrophage migration requires MMP-9 activation by plasminogen in miceJCI Insight, 2008
- Unique and redundant roles of α4 and β2 integrins in kinetics of recruitment of lymphoid vs myeloid cell subsets to the inflamed peritoneum revealed by studies of genetically deficient miceExperimental Hematology, 2007
- Endocytic receptor LRP together with tPA and PAI-1 coordinates Mac-1-dependent macrophage migrationThe EMBO Journal, 2006
- An Essential Role for Ectodomain Shedding in Mammalian DevelopmentScience, 1998