Dissociation between early and late events in T cell activation mediated through CD28 surface molecule
- 31 May 1991
- journal article
- Published by Elsevier BV in Molecular Immunology
- Vol. 28 (4-5), 427-435
- https://doi.org/10.1016/0161-5890(91)90156-e
Abstract
No abstract availableKeywords
This publication has 24 references indexed in Scilit:
- Human T cell activation: differential response to anti‐CD28 as compared to anti‐CD3 monoclonal antibodiesEuropean Journal of Immunology, 1989
- T cell activation via the CD2 molecule is associated with protein kinase C translocation from the cytosol to the plasma membraneEuropean Journal of Immunology, 1989
- Regulation of Lymphokine Messenger RNA Stability by a Surface-Mediated T Cell Activation PathwayScience, 1989
- Correlation of CD2 binding and functional properties of multimeric and monomeric lymphocyte function-associated antigen 3.The Journal of Experimental Medicine, 1989
- Transmembrane signalling via the T11‐dependent pathway of human T cell activation. Evidence for the involvement of 1,2‐diacylglycerol and inositol phosphatesEuropean Journal of Immunology, 1987
- Anti‐CD2 (sheep red blood cell receptor) monoclonal antibodies and T cell activation I. Pairs of anti‐T11.1 and T11.2 (CD2 subgroups) are strongly mitogenic for T cells in presence of 12‐O‐tetradecanoylphorbol 13‐acetateEuropean Journal of Immunology, 1986
- Signal transducing mechanisms involved in human T cell activation via surface T44 molecules. Comparison with signals transduced via the T cell receptor complexEuropean Journal of Immunology, 1986
- Involvement of T44 molecules in an antigen-independent pathway of T cell activation. Analysis of the correlations to the T cell antigen-receptor complex.The Journal of Experimental Medicine, 1985
- Human T cell activation. II. A new activation pathway used by a major T cell population via a disulfide-bonded dimer of a 44 kilodalton polypeptide (9.3 antigen).The Journal of Experimental Medicine, 1985
- Transmembrane signalling by the T cell antigen receptor. Perturbation of the T3-antigen receptor complex generates inositol phosphates and releases calcium ions from intracellular stores.The Journal of Experimental Medicine, 1985