Influence of liver dysfunction on volume of putaminal hemorrhage.
- 1 August 1988
- journal article
- abstracts
- Published by Ovid Technologies (Wolters Kluwer Health) in Stroke
- Vol. 19 (8), 987-990
- https://doi.org/10.1161/01.str.19.8.987
Abstract
We studied the relations of age, sex, hypertension, alcohol consumption, liver dysfunction, and thrombocyte count to the volume of the hematoma in 141 patients with spontaneous putaminal hemorrhage. Hematomas were significantly larger in men, regular alcohol consumers, those with liver dysfunction, and those with low platelet counts. Our findings reflect the fact that almost all of the alcohol consumers were men, most of them had liver disorders, and the volume of hematoma in such patients was relatively large.This publication has 15 references indexed in Scilit:
- Stroke and Alcohol ConsumptionThe New England Journal of Medicine, 1986
- PRESSOR EFFECT OF ALCOHOL IN HYPERTENSIONThe Lancet, 1984
- Hepatic cirrhosis and intracranial hemorrhage: Significance of the association in 53 pathological casesAnnals of Neurology, 1980
- The decreasing incidence of primary intracerebral hemorrhage: A population studyAnnals of Neurology, 1979
- Alcohol Consumption and Blood PressureThe New England Journal of Medicine, 1977
- Fatal hypertensive intracerebral haematomas: a survey of the pathological anatomy of 393 cases.Journal of Neurology, Neurosurgery & Psychiatry, 1968
- The occurrence and significance of intracerebral micro‐aneurysmsThe Journal of Pathology and Bacteriology, 1967
- Suppression of Hematopoiesis by Ethanol *JCI Insight, 1964
- Epidemiological Studies on Cerebrovascular Diseases in Hisayama, Kyushu Island, JapanJapanese Heart Journal, 1964
- OBSERVATIONS ON INTRACEREBRAL ANEURYSMSBrain, 1963