Brief Secondhand Smoke Exposure Depresses Endothelial Progenitor Cells Activity and Endothelial Function: Sustained Vascular Injury and Blunted Nitric Oxide Production
- 6 May 2008
- journal article
- Published by Elsevier BV in Journal of the American College of Cardiology
- Vol. 51 (18), 1760-1771
- https://doi.org/10.1016/j.jacc.2008.01.040
Abstract
No abstract availableThis publication has 47 references indexed in Scilit:
- Declines in Hospital Admissions for Acute Myocardial Infarction in New York State After Implementation of a Comprehensive Smoking BanAmerican Journal of Public Health, 2007
- Pleiotrophin induces nitric oxide dependent migration of endothelial progenitor cellsJournal of Cellular Physiology, 2007
- Acute smoke-induced endothelial dysfunction is more prolonged in smokers than in non-smokersInternational Journal of Cardiology, 2007
- Chimney Stove Intervention to Reduce Long-term Wood Smoke Exposure Lowers Blood Pressure among Guatemalan WomenEnvironmental Health Perspectives, 2007
- Circulating MicroparticlesHypertension, 2006
- Soluble vascular endothelial growth factor, soluble VEGF receptor Flt-1 and endothelial function in healthy smokersInternational Journal of Cardiology, 2005
- Endogenous nitric oxide activation protects against cigarette smoking induced apoptosis in endothelial cellsFEBS Letters, 2005
- The pathophysiology of cigarette smoking and cardiovascular disease: An updateJournal of the American College of Cardiology, 2004
- Red wine's antioxidants counteract acute endothelial dysfunction caused by cigarette smoking in healthy nonsmokersAmerican Heart Journal, 2004
- Effects of l -Arginine on Atherogenesis and Endothelial Dysfunction due to Secondhand SmokeHypertension, 1999