17β-Estradiol normalizes Toll receptor 4, mitogen activated protein kinases and inflammatory response in epidermal keratinocytes following trauma-hemorrhage
- 31 July 2007
- journal article
- Published by Elsevier BV in Molecular Immunology
- Vol. 44 (13), 3317-3323
- https://doi.org/10.1016/j.molimm.2007.02.023
Abstract
No abstract availableKeywords
This publication has 30 references indexed in Scilit:
- 17β-Estradiol downregulates Kupffer cell TLR4-dependent p38 MAPK pathway and normalizes inflammatory cytokine production following trauma-hemorrhageMolecular Immunology, 2007
- The role of MAPK in Kupffer cell toll-like receptor (TLR) 2-, TLR4-, and TLR9-mediated signaling following trauma-hemorrhageJournal of Cellular Physiology, 2006
- Estrogen increases protective proteins following trauma and hemorrhageAmerican Journal of Physiology-Regulatory, Integrative and Comparative Physiology, 2006
- Tissue-specific expression of estrogen receptors and their role in the regulation of neutrophil infiltration in various organs following trauma-hemorrhageJournal of Leukocyte Biology, 2006
- A role of PP1/PP2A in mesenteric lymph node T cell suppression in a two-hit rodent model of alcohol intoxication and injuryJournal of Leukocyte Biology, 2005
- Interaction Between the Innate and Adaptive Immune Systems is Required to Survive Sepsis and Control Inflammation After InjuryShock, 2003
- Keratinocytes and cytokinesVeterinary Immunology and Immunopathology, 2002
- TRAUMA-INDUCED SUPPRESSION OF ANTIGEN PRESENTATION AND EXPRESSION OF MAJOR HISTOCOMPATIBILITY CLASS II ANTIGEN COMPLEX IN LEUKOCYTESShock, 1996
- A MAP Kinase Targeted by Endotoxin and Hyperosmolarity in Mammalian CellsScience, 1994
- Keratinocytes as initiators of inflammationThe Lancet, 1991