Redox-fibrosis: Impact of TGFβ1 on ROS generators, mediators and functional consequences
Open Access
- 1 December 2015
- journal article
- review article
- Published by Elsevier BV in Redox Biology
- Vol. 6, 344-352
- https://doi.org/10.1016/j.redox.2015.08.015
Abstract
No abstract availableKeywords
Funding Information
- Biocenter Oulu, the Academy of Finland (251314, 2012–2017)
- Sigrid Juselius Foundation
- European Cooperation in Science and Technology (BM1203/EU‐ROS)
This publication has 161 references indexed in Scilit:
- Variation in selenoenzyme genes and prostate cancer risk and survivalThe Prostate, 2012
- MeCP2 controls the expression of RASAL1 in the hepatic fibrosis in ratsToxicology, 2011
- The nicotinamide adenine dinucleotide phosphate oxidase (NOX) homologues NOX1 and NOX2/gp91phox mediate hepatic fibrosis in miceHepatology, 2011
- Aging, antagonistic pleiotropy and fibrotic diseaseThe International Journal of Biochemistry & Cell Biology, 2010
- Nox enzymes from fungus to fly to fish and what they tell us about Nox function in mammalsFree Radical Biology & Medicine, 2010
- Enhanced expression of Nrf2 in mice attenuates the fatty liver produced by a methionine- and choline-deficient dietToxicology and Applied Pharmacology, 2010
- Nox proteins in signal transductionFree Radical Biology & Medicine, 2009
- Deficiency of nicotinamide adenine dinucleotide phosphate, reduced form oxidase enhances hepatocellular injury but attenuates fibrosis after chronic carbon tetrachloride administrationHepatology, 2008
- NOX in liver fibrosisArchives of Biochemistry and Biophysics, 2007
- Mortality in Randomized Trials of Antioxidant Supplements for Primary and Secondary PreventionJAMA, 2007