Normal osteoclast number and function in rat pups lacking parathyroid hormone
- 1 July 1980
- journal article
- research article
- Published by Springer Science and Business Media LLC in Cellular and Molecular Life Sciences
- Vol. 36 (7), 871-872
- https://doi.org/10.1007/bf01978622
Abstract
In parathyroidectomized suckling rats, bone modeling and the number and activity of cells in the osteoclast population are normal. These findings are at variance with observations in older animals and suggest that factors other than parathyroid hormone influence osteoclast formation and function in the neonate.This publication has 21 references indexed in Scilit:
- Effects of parathyroid hormone on the osteoclastic pool, bone resorption and formation in rat alveolar boneCalcified Tissue International, 1978
- Resorbing bone is chemotactic for monocytesNature, 1978
- Prostaglandin and thromboxane synthesis in a pure macrophage population and the inhibition, by E‐type prostaglandins, of chemiluminescenceFEBS Letters, 1978
- Contact-Mediated Bone Resorption by Human Monocytes in VitroScience, 1978
- THE EFFECTS OF PARATHYROID HORMONE, COLCHICINE, AND CALCITONIN ON THE ULTRASTRUCTURE AND THE ACTIVITY OF OSTEOCLASTS IN ORGAN CULTUREThe Journal of cell biology, 1974
- Calcitonin and Parathyroid Hormone – Relation to Early Neonatal Hypocalcemia in Infants of Diabetic MothersNeonatology, 1974
- Neonatal parathyroid function: Role of gestational age and postnatal ageThe Journal of Pediatrics, 1973
- Inhibition of Bone Matrix Formation, Mineralization, and Resorption in Thyroparathyroidectomized RatsJCI Insight, 1973
- Bone Resorbing Activity in Supernatant Fluid from Cultured Human Peripheral Blood LeukocytesScience, 1972
- ON THE MECHANISMS OF BONE RESORPTIONThe Journal of cell biology, 1968