NOX4-driven ROS formation mediates PTP inactivation and cell transformation in FLT3ITD-positive AML cells
- 26 August 2015
- journal article
- research article
- Published by Springer Science and Business Media LLC in Leukemia
- Vol. 30 (2), 473-483
- https://doi.org/10.1038/leu.2015.234
Abstract
No abstract availableKeywords
This publication has 61 references indexed in Scilit:
- Genomic and Epigenomic Landscapes of Adult De Novo Acute Myeloid LeukemiaNew England Journal of Medicine, 2013
- Nox4-dependent ROS modulation by amino endoperoxides to induce apoptosis in cancer cellsCell Death & Disease, 2013
- Validation of ITD mutations in FLT3 as a therapeutic target in human acute myeloid leukaemiaNature, 2012
- Genetic and Pharmacologic Inhibition of β-Catenin Targets Imatinib-Resistant Leukemia Stem Cells in CMLCell Stem Cell, 2012
- Global Proteomic Assessment of the Classical Protein-Tyrosine Phosphatome and “Redoxome”Cell, 2011
- Signal transduction by reactive oxygen speciesThe Journal of cell biology, 2011
- Protein-tyrosine Phosphatase DEP-1 Controls Receptor Tyrosine Kinase FLT3 SignalingJournal of Biological Chemistry, 2011
- NADPH oxidase-4 mediates protection against chronic load-induced stress in mouse hearts by enhancing angiogenesisProceedings of the National Academy of Sciences, 2010
- NADPH oxidase-4 mediates myofibroblast activation and fibrogenic responses to lung injuryNature Medicine, 2009
- MLL-AF9 and FLT3 cooperation in acute myelogenous leukemia: development of a model for rapid therapeutic assessmentLeukemia, 2007