Interplay between bax, reactive oxygen species production, and cardiolipin oxidation during apoptosis
- 28 March 2008
- journal article
- research article
- Published by Elsevier BV in Biochemical and Biophysical Research Communications
- Vol. 368 (1), 145-150
- https://doi.org/10.1016/j.bbrc.2008.01.055
Abstract
No abstract availableKeywords
This publication has 28 references indexed in Scilit:
- A Mitochondria-Targeted Nitroxide/Hemigramicidin S Conjugate Protects Mouse Embryonic Cells Against Gamma IrradiationInternational Journal of Radiation Oncology*Biology*Physics, 2008
- The Hierarchy of Structural Transitions Induced in Cytochrome c by Anionic Phospholipids Determines Its Peroxidase Activation and Selective Peroxidation during Apoptosis in CellsBiochemistry, 2007
- Cardiolipin: Setting the beat of apoptosisApoptosis, 2007
- Structural Requirements for Optimized Delivery, Inhibition of Oxidative Stress, and Antiapoptotic Activity of Targeted NitroxidesJournal of Pharmacology and Experimental Therapeutics, 2006
- Early changes in intramitochondrial cardiolipin distribution during apoptosis.2002
- A Bax-Induced Pro-Oxidant State Is Critical for CytochromecRelease during Programmed Neuronal DeathJournal of Neuroscience, 2002
- Cytochrome c release from mitochondria proceeds by a two-step processProceedings of the National Academy of Sciences of the United States of America, 2002
- Decreased Cardiolipin Synthesis Corresponds with Cytochromec Release in Palmitate-induced Cardiomyocyte ApoptosisJournal of Biological Chemistry, 2001
- Unfolding and refolding of cytochrome c driven by the interaction with lipid micellesProtein Science, 2000
- Loss of Molecular Interaction between Cytochrome c and Cardiolipin Due to Lipid PeroxidationBiochemical and Biophysical Research Communications, 1999