Loss of p16/INK4A Protein Expression in Non-Hodgkin's Lymphomas Is a Frequent Finding Associated with Tumor Progression
- 30 September 1998
- journal article
- Published by Elsevier BV in The American Journal of Pathology
- Vol. 153 (3), 887-897
- https://doi.org/10.1016/s0002-9440(10)65630-1
Abstract
No abstract availableKeywords
This publication has 47 references indexed in Scilit:
- Hypermethylation of a 5′ CpG island of p16 is a frequent event in non-Hodgkin’s lymphomaLeukemia, 1997
- Frequency of homozygous deletion at p16/CDKN2 in primary human tumoursNature Genetics, 1995
- Homozygous loss of the MTSl/pl6 and MTS2/pl5 genes in lymphoma and lymphoblastic leukaemia cell linesBritish Journal of Haematology, 1995
- Inhibitors of mammalian G1 cyclin-dependent kinases.Genes & Development, 1995
- Joining the complex: Cyclin-dependent kinase inhibitory proteins and the cell cycleCell, 1994
- pl5INK4B is a potentia| effector of TGF-β-induced cell cycle arrestNature, 1994
- A Cell Cycle Regulator Potentially Involved in Genesis of Many Tumor TypesScience, 1994
- Deletions of the cyclin-dependent kinase-4 inhibitor gene in multiple human cancersNature, 1994
- A new regulatory motif in cell-cycle control causing specific inhibition of cyclin D/CDK4Nature, 1993
- Flow cytometric detection of the mitochondrial BCL‐2 protein in normal and neoplastic human lymphoid cellsCytometry, 1992