Changes in Hypothalamic Agouti-Related Protein (AGRP), but not α-MSH or Pro-Opiomelanocortin Concentrations in Dietary-Obese and Food-Restricted Rats
- 19 May 1999
- journal article
- Published by Elsevier BV in Biochemical and Biophysical Research Communications
- Vol. 258 (3), 574-577
- https://doi.org/10.1006/bbrc.1999.0675
Abstract
No abstract availableKeywords
This publication has 21 references indexed in Scilit:
- Characterization of the Neuroanatomical Distribution of Agouti-Related Protein Immunoreactivity in the Rhesus Monkey and the Rat*Endocrinology, 1999
- Altered energy balance causes selective changes in melanocortin-4(MC4-R), but not melanocortin-3 (MC3-R), receptors in specific hypothalamic regions: further evidence that activation of MC4-R is a physiological inhibitor of feeding.Diabetes, 1999
- The neuropeptide Y/agouti gene-related protein (AGRP) brain circuitry in normal, anorectic, and monosodium glutamate-treated miceProceedings of the National Academy of Sciences of the United States of America, 1998
- A frameshift mutation in MC4R associated with dominantly inherited human obesityNature Genetics, 1998
- Proopiomelanocortin Neurons Are Direct Targets for Leptin in the HypothalamusEndocrinology, 1997
- Differential Agonist Regulation of the Human κ‐Opioid ReceptorJournal of Neurochemistry, 1997
- Hypothalamic expression of ART, a novel gene related to agouti, is up-regulated in obese and diabetic mutant mice.Genes & Development, 1997
- Residues Specifically Involved in Down‐Regulation but Not Internalization of the m1 Muscarinic Acetylcholine ReceptorJournal of Neurochemistry, 1997
- Targeted Disruption of the Melanocortin-4 Receptor Results in Obesity in MiceCell, 1997
- Role of melanocortinergic neurons in feeding and the agouti obesity syndromeNature, 1997